G Protein-Coupled Receptor Signaling in Metabolic Disease

G protein-coupled receptors (GPCRs) are the most abundant receptor family encoded by the human genome and are common targets for the treatment of multiple diseases. GPCRs are involved in regulating many aspects of cellular physiology and metabolism and recently have been targeted for the treatment of metabolic disorders, such as Type 2 diabetes. The goal of this Call is to highlight the role of GPCR signaling in metabolic disease and GPCRs as targets for the treatment of obesity and diabetes and their associated comorbidities. Topics within the scope of this Call include but are not limited to: 

  1. Nontraditional GPCR signaling and its impact on metabolism.

  2. The roles of GPCR signaling in the control of food intake, gastric emptying, pancreatic hormone release, insulin sensitivity, and energy expenditure.

  3. Targeting GPCRs to reduce appetite or improve insulin sensitivity or release.

  4. Interaction of GPCRs with tyrosine kinase receptors.

  5. Expression and function of GPCRs following gastric bypass surgery.

  6. Novel methods to explore GPCR signaling and function.

All manuscripts should be submitted online via eJournal Press. During the online submission process, under the “Keywords & Special Sections” tab, please use the “Category” drop-down menu and select “Call for Papers: GPCR Signaling”.

Manuscripts will undergo normal peer review as they are received. Accepted manuscripts will be published online as they are accepted. Articles published from this Call for Papers will be highlighted at the time of publication. The collection also may include specially commissioned review articles related to the topic. Dr. Gina Yosten (Saint Louis University) will serve as the Guest Editor for this Call.

Manuscripts can be submitted anytime, but to be eligible for inclusion in this Call for Papers, manuscripts must be submitted by December 31, 2017.


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